Effects of acute exposure to mercury chloride and methylmercury on plasma cortisol, T3, T4, glucose and liver glycogen in rainbow trout (Oncorhynchus mykiss)

Our results indicate that both mercurial compounds stimulate the pituitary-interrenal and the pituitary-thyroid axis and modify the carbohydrate metabolism in juvenile rainbow trout, and that the organic mercury CH3Hg+ is a more potent chemical stressor than the inorganic Hg2+

H. Bleau; C. Daniel; G. Chevalier; H. van Tra; A. Hontela

2002

Scholarcy highlights

  • Juvenile rainbow trout were exposed to mercury chloride HgCl2, and to methylmercury chloride CH3HgCl for 4, 72 and 168 h to determine the effects of sublethal doses of these compounds on the hypothalamo-pituitary-interrenal and the hypothalamo-pituitary-thyroid axes
  • A decrease in liver glycogen reserves was detected after 1 week of exposure to 6 μg/l CH3HgCl
  • Our results indicate that both mercurial compounds stimulate the pituitary-interrenal and the pituitary-thyroid axis and modify the carbohydrate metabolism in juvenile rainbow trout, and that the organic mercury CH3Hg+ is a more potent chemical stressor than the inorganic Hg2+

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